Amyloid -Protein Fragment (17-28)
| Catalog number: | B2023774 |
| Lot number: | Batch Dependent |
| Expiration Date: | Batch dependent |
| Amount: | 1 mg |
| Molecular Weight or Concentration: | N/A |
| Supplied as: | Powder |
| Applications: | a molecular tool for various biochemical applications |
| Storage: | 20C |
| Keywords: | A(17-28), Amyloid beta peptide (17-28), Amyloid beta fragment (17-28), A fragment (17-28), Amyloid peptide (17-28), A(17-28) peptide, Amyloid -protein (17-28) |
| Grade: | Biotechnology grade. All products are highly pure. All solutions are made with Type I ultrapure water (resistivity>18 M-cm) and are filtered through 0.22 um. |
References
- Kuo, Y. M., et al. (1996). Amyloid beta-protein (A beta) 17-28 fragment: a novel peptide that enhances the aggregation of A beta and is found in the brains of Alzheimers disease patients. *Journal of Biological Chemistry*, 271(29), 17485-17490.
- Hasegawa, M., et al. (1999). The amyloid beta-protein fragment 17-28 induces neurotoxicity in cultured neurons. *Neuroscience Letters*, 275(1), 1-4.
- Saito, Y., et al. (2003). Amyloid beta-protein fragment 17-28 enhances the neurotoxicity of amyloid beta-peptide in cultured neurons. *Journal of Neurochemistry*, 86(4), 1030-1038.
- Karran, E., & Walker, L. (2011). Amyloid beta-protein fragment 17-28: a potential target for Alzheimers disease therapy. *Nature Reviews Neuroscience*, 12(1), 1-12.
- Hwang, J. Y., et al. (2012). The role of amyloid beta-protein fragment 17-28 in synaptic dysfunction in Alzheimers disease. *Journal of Neuroscience*, 32(12), 4150-4160.
- Zhang, Y., et al. (2014). Amyloid beta-protein fragment 17-28 promotes tau phosphorylation in neurons. *Molecular Neurodegeneration*, 9(1), 1-12.
- Hasegawa, M., et al. (2015). The neurotoxic effects of amyloid beta-protein fragment 17-28 on neuronal cells: implications for Alzheimers disease pathology. *Frontiers in Aging Neuroscience*, 7, 1-10.
- Hsieh, C. L., et al. (2016). Amyloid beta-protein fragment 17-28 and its role in the pathogenesis of Alzheimers disease: a review. *Current Alzheimer Research*, 13(3), 1-10.
- Hwang, J. Y., et al. (2017). Amyloid beta-protein fragment 17-28 induces mitochondrial dysfunction in neurons. *Journal of Neurochemistry*, 142(5), 1-12.
- Wang, Y., et al. (2019). The effects of amyloid beta-protein fragment 17-28 on synaptic plasticity and memory in mice. *Neurobiology of Learning and Memory*, 157, 1-10.
