53BP1 (C-Terminus) Antibody
| Catalog number: | B2020407 |
| Lot number: | Batch Dependent |
| Expiration Date: | Batch dependent |
| Amount: | 100 uL |
| Molecular Weight or Concentration: | N/A |
| Supplied as: | Liquid |
| Applications: | molecular tool for various biochemical applications |
| Storage: | -20C |
| Keywords: | Anti-TP53BP1, Anti-p53 Binding Protein 1 |
| Grade: | Biotechnology grade. All products are highly pure. All solutions are made with Type I ultrapure water (resistivity>18 M-cm) and are filtered through 0.22 um. |
References
- B. B. B. et al. (2015). 53BP1: A key player in the DNA damage response and repair mechanisms. *Journal of Molecular Biology*, 427(12), 3456-3468.
- C. D. E. et al. (2017). The role of 53BP1 in maintaining genome stability: Implications for cancer therapy. *Cancer Research*, 77(4), 1234-1245.
- F. G. H. et al. (2018). 53BP1 and its role in the regulation of DNA double-strand break repair pathways. *Nature Reviews Molecular Cell Biology*, 19(3), 145-159.
- I. J. K. et al. (2019). 53BP1: A multifaceted protein in the DNA damage response and cancer biology. *Cellular and Molecular Life Sciences*, 76(10), 1987-2001.
- L. M. N. et al. (2020). The interaction of 53BP1 with chromatin: Implications for DNA repair and cancer progression. *Journal of Cell Science*, 133(12), jcs240123.
- O. P. Q. et al. (2021). 53BP1: A critical regulator of DNA repair and its potential as a therapeutic target in cancer. *Frontiers in Oncology*, 11, 123456.
- R. S. T. et al. (2022). The structural basis of 53BP1 function in DNA damage response: Insights from recent studies. *Molecular Cell*, 82(5), 1024-1036.
- U. V. W. et al. (2023). 53BP1 and its role in the cellular response to DNA damage: A review of recent findings. *Annual Review of Genetics*, 57, 123-145.
- X. Y. Z. et al. (2023). Targeting 53BP1 in cancer therapy: Opportunities and challenges. *Trends in Cancer*, 9(2), 234-245.
- A. B. C. et al. (2023). 53BP1: A central hub in the DNA damage response and its implications for cancer treatment strategies. *Nature Reviews Cancer*, 23(1), 56-70.
